Ozone-induced caspase-like activities are dependent on early ion channel regulations and ROS generation in Arabidopsis thaliana cells.

Using A. thaliana cultured cells; we recently reported new insights regarding the effect of acute O 3 exposure. ( 1) (-) ( 3) This consist in an oxidative dependent controlled cell death process involving cell shrinkage due to an early activation of anion channel ( 1) and a delayed activation of K (+) outward currents ( 2) (,) but also to early events like Ca ( 2+) influx or singlet oxygen production possibly linked to mitochondrial dysfunction. ( 1) Here we provide evidence that most of these early events act downstream of caspase-like activities as recently demonstrated for K (+) channel activation. ( 2).