Host specific T-cell response is critical for the outcome of Helicobacter pylori infection. In genetically susceptible individuals, H. pylori can activate gastric CD4+ Th1 cells that recognize cross-reactive epitopes shared by H. pylori proteins and self H+, K+-ATPase, leading to gastric autoimmunity via molecular mimicry.

Helicobacter pylori and gastric autoimmunity / MM. D'ELIOS MM; MP. BERGMAN; A. AMEDEI; BJ. APPELMELK; G. DEL PRETE. - In: MICROBES AND INFECTION. - ISSN 1286-4579. - STAMPA. - 6:(2004), pp. 1395-1401. [10.1016/j.micinf.2004.10.001]

Helicobacter pylori and gastric autoimmunity.

D'ELIOS, MARIO MILCO;AMEDEI, AMEDEO;DEL PRETE, GIANFRANCO
2004

Abstract

Host specific T-cell response is critical for the outcome of Helicobacter pylori infection. In genetically susceptible individuals, H. pylori can activate gastric CD4+ Th1 cells that recognize cross-reactive epitopes shared by H. pylori proteins and self H+, K+-ATPase, leading to gastric autoimmunity via molecular mimicry.
2004
6
1395
1401
MM. D'ELIOS MM; MP. BERGMAN; A. AMEDEI; BJ. APPELMELK; G. DEL PRETE
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Utilizza questo identificatore per citare o creare un link a questa risorsa: https://hdl.handle.net/2158/308927
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