In chronic hypoperfusion settings, such as chronic heart failure (CHF), bowel dysfunction and a related higher intestinal permeability have already been demonstrated. It has been reported that in CHF, cardiac output decreases and CHF-associated gut mucosal edema compromises gut function, leading to the translocation of endotoxin and gut bacteria [1] and [2]. There is a lack of data about bowel permeability behavior in an acute hypoperfusion setting, as cardiogenic shock (CS) following ST elevation myocardial infarction (STEMI).

Endotoxin role in cardiogenic shock: A brief report / Attanà P;Lazzeri C;Chiostri M;Picariello C;Gensini GF;Valente S. - In: INTERNATIONAL JOURNAL OF CARDIOLOGY. - ISSN 0167-5273. - STAMPA. - 167:(2013), pp. 3031-3032. [10.1016/j.ijcard.2012.11.100]

Endotoxin role in cardiogenic shock: A brief report.

CHIOSTRI, MARCO;PICARIELLO, CLAUDIO;GENSINI, GIAN FRANCO;
2013

Abstract

In chronic hypoperfusion settings, such as chronic heart failure (CHF), bowel dysfunction and a related higher intestinal permeability have already been demonstrated. It has been reported that in CHF, cardiac output decreases and CHF-associated gut mucosal edema compromises gut function, leading to the translocation of endotoxin and gut bacteria [1] and [2]. There is a lack of data about bowel permeability behavior in an acute hypoperfusion setting, as cardiogenic shock (CS) following ST elevation myocardial infarction (STEMI).
2013
167
3031
3032
Attanà P;Lazzeri C;Chiostri M;Picariello C;Gensini GF;Valente S
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Utilizza questo identificatore per citare o creare un link a questa risorsa: https://hdl.handle.net/2158/785532
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