in patients with flow-limiting vascular disease, ischemic mediators may cause maximal compensatory vasodilation. In such cases, NO released from SNO-Hb in peripheral capillaries would not lead to further dilatation, but could still offer significant vasculoprotection through anti-platelet, anti-inflammatory, insulin-sensitizing, and progenitor-cell stimulating effects
Last word on viewpoint: Anemia contributes to cardiovascular disease through reductions in nitric oxide / Andreotti, F., Coluzzi, G., Pafundi, T., Rio, T., Navarese, E.P., Crea, F., Pistolesi, M., Maseri, A., Hennekens, C.H.. - In: JOURNAL OF APPLIED PHYSIOLOGY. - ISSN 8750-7587. - CD-ROM. - 122:(2017), pp. 420-421. [10.1152/japplphysiol.01095.2016]
Last word on viewpoint: Anemia contributes to cardiovascular disease through reductions in nitric oxide
Pistolesi, Massimo;
2017
Abstract
in patients with flow-limiting vascular disease, ischemic mediators may cause maximal compensatory vasodilation. In such cases, NO released from SNO-Hb in peripheral capillaries would not lead to further dilatation, but could still offer significant vasculoprotection through anti-platelet, anti-inflammatory, insulin-sensitizing, and progenitor-cell stimulating effects
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