ROVIDA, ELISABETTA
 Distribuzione geografica
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Continente #
NA - Nord America 8.438
EU - Europa 6.708
AS - Asia 3.369
SA - Sud America 516
AF - Africa 72
OC - Oceania 57
Continente sconosciuto - Info sul continente non disponibili 3
Totale 19.163
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Nazione #
US - Stati Uniti d'America 8.335
PL - Polonia 2.143
RU - Federazione Russa 2.063
SG - Singapore 934
CN - Cina 766
IT - Italia 706
HK - Hong Kong 621
SE - Svezia 482
IE - Irlanda 477
BR - Brasile 407
KR - Corea 309
VN - Vietnam 218
DE - Germania 209
IN - India 165
FI - Finlandia 142
GB - Regno Unito 138
JO - Giordania 134
FR - Francia 104
UA - Ucraina 76
CA - Canada 58
ID - Indonesia 57
AU - Australia 56
AR - Argentina 41
NL - Olanda 41
CH - Svizzera 38
JP - Giappone 30
CI - Costa d'Avorio 29
TR - Turchia 26
MX - Messico 25
BE - Belgio 22
EC - Ecuador 22
TW - Taiwan 17
AT - Austria 14
IQ - Iraq 13
BD - Bangladesh 12
VE - Venezuela 12
ZA - Sudafrica 12
CO - Colombia 11
ES - Italia 11
MA - Marocco 10
MK - Macedonia 10
PH - Filippine 8
LK - Sri Lanka 7
PE - Perù 7
AE - Emirati Arabi Uniti 6
IR - Iran 6
JM - Giamaica 6
RO - Romania 6
SA - Arabia Saudita 6
CL - Cile 5
IL - Israele 5
LT - Lituania 5
PK - Pakistan 5
PY - Paraguay 5
BJ - Benin 4
EG - Egitto 4
KE - Kenya 4
MY - Malesia 4
AL - Albania 3
BO - Bolivia 3
CR - Costa Rica 3
DO - Repubblica Dominicana 3
DZ - Algeria 3
HU - Ungheria 3
KG - Kirghizistan 3
KW - Kuwait 3
TH - Thailandia 3
UY - Uruguay 3
AM - Armenia 2
AZ - Azerbaigian 2
BB - Barbados 2
BG - Bulgaria 2
EE - Estonia 2
EU - Europa 2
NO - Norvegia 2
PA - Panama 2
RS - Serbia 2
TN - Tunisia 2
UZ - Uzbekistan 2
BA - Bosnia-Erzegovina 1
BN - Brunei Darussalam 1
BW - Botswana 1
BY - Bielorussia 1
CZ - Repubblica Ceca 1
DK - Danimarca 1
ET - Etiopia 1
GP - Guadalupe 1
HN - Honduras 1
IM - Isola di Man 1
LB - Libano 1
LU - Lussemburgo 1
LV - Lettonia 1
LY - Libia 1
MM - Myanmar 1
NG - Nigeria 1
NP - Nepal 1
NZ - Nuova Zelanda 1
OM - Oman 1
PR - Porto Rico 1
TT - Trinidad e Tobago 1
Totale 19.162
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Città #
Warsaw 2.141
Santa Clara 2.078
Ashburn 840
Fairfield 754
Singapore 646
Chandler 538
Hong Kong 518
Dublin 472
Woodbridge 370
Seattle 333
Seoul 301
Cambridge 290
Houston 269
Florence 258
Beijing 246
Wilmington 243
Ann Arbor 205
Jacksonville 149
Hefei 126
Princeton 119
Buffalo 110
Lawrence 110
Altamura 104
Los Angeles 95
Boston 93
Munich 88
Dallas 72
Dong Ket 72
Mumbai 68
Moscow 67
Boardman 66
Paris 66
Shanghai 62
San Diego 58
Kent 57
Melbourne 55
Jakarta 44
Ho Chi Minh City 42
Medford 40
Falls Church 39
Milan 38
Pune 34
São Paulo 34
Turku 31
The Dalles 30
Abidjan 29
New York 29
Bern 28
Redondo Beach 28
Hanoi 25
Rome 25
Frankfurt am Main 24
London 24
Brussels 22
Clifton 22
Phoenix 22
Norwalk 21
Portsmouth 21
Tokyo 21
Toronto 20
Ottawa 19
Council Bluffs 16
Helsinki 16
Lappeenranta 15
Yubileyny 15
Columbus 14
Redwood City 14
Rio de Janeiro 13
Siena 13
Brooklyn 12
Daan 12
Dearborn 12
Guangzhou 12
Lauterbourg 12
Orem 12
Andover 11
Chicago 11
Hangzhou 11
Stockholm 11
Vienna 11
Chennai 10
Montreal 10
Goiânia 9
Belo Horizonte 8
Brasília 8
Colombo 8
Da Nang 8
Guayaquil 8
Prato 8
Atlanta 7
Bengaluru 7
Guarulhos 7
Hillsboro 7
Hyderabad 7
Nuremberg 7
Quito 7
Redmond 7
Tianjin 7
Turin 7
Barcelona 6
Totale 13.247
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Nome #
One more stem cell niche: how the sensitivity of chronic myeloid leukemia cells to imatinib mesylate is modulated within a “hypoxic” environment 359
The Colony-Stimulating Factor-1 (CSF-1) Receptor Sustains ERK1/2 Activation and Proliferation in Breast Cancer Cell Lines. 353
The Leukemic Stem Cell Niche: Adaptation to “Hypoxia” versus Oncogene Addiction 323
The metabolically-modulated stem cell niche: a dynamic scenario regulating cancer cell phenotype and resistance to therapy. 322
Targeting chronic myeloid leukemia stem cells with the hypoxia-inducible factor inhibitor acriflavine. 317
Chromatin-associated CSF-1R binds to the promoter of proliferation-related genes in breast cancer cells. 313
Hypoxia-resistant profile implies vulnerability of cancer stem cells to physiological agents, which suggests new therapeutic targets. 307
ERK5 is activated by oncogenic BRAF and promotes melanoma growth 306
The complex metabolic network gearing the G1/S transition in leukemic stem cells: Hints to a rational use of antineoplastic agents. 304
Colony-Stimulating Factor-1 Receptor in the Polarization of Macrophages: A Target for Turning Bad to Good Ones? 302
Chronic myeloid leukemia and hepatoblastoma: Two cancer models to link metabolism to stem cells 296
Hypoxia selects bortezomib-resistant stem cells of chronic myeloid leukemia. 295
The expansion of murine bone marrow cells preincubated in hypoxia as an in vitro indicator of their marrow-repopulating ability. 268
Hypoxia up-regulates SERPINB3 through HIF-2α in human liver cancer cells. 266
Targeting the Extracellular Signal-Regulated Kinase 5 Pathway to Suppress Human Chronic Myeloid Leukemia Stem Cells 262
Aberrant Glycosphingolipid Pattern in Human Cholangiocarcinoma Stem-Like Subsets 261
The mitogen-activated protein kinase ERK5 regulates the development and growth of hepatocellular carcinoma. 244
Impact of ERK5 on the Hallmarks of Cancer 234
1,25-dihydroxyvitamin D3 and erk ½ map kinases in a human chronic myeloid leukemia cell line 226
Low-dose methotrexate enhances cycling of highly anaplastic cancer cells. 224
Beyond Kinase Activity: ERK5 Nucleo-Cytoplasmic Shuttling as a Novel Target for Anticancer Therapy 216
Different BCR/Abl protein suppression patterns as a converging trait of chronic myeloid leukemia cell adaptation to energy restriction 210
Fibronectin induces macrophage migration through a SFK-FAK/CSF-1R pathway 208
The protein kinase CK2 contributes to the malignant phenotype of cholangiocarcinoma cells 207
Anti-β4 integrin autoantibodies in patients with mucous membrane pemphigoid: a retrospective analysis from a tertiary centre in Italy 204
In Vitro Comparison of the Effects of Imatinib and Ponatinib on Chronic Myeloid Leukemia Progenitor/Stem Cell Features 195
HYPOXIA SUPPRESSES BCR/ABL AND SELECTS IMATINIB-INSENSITIVE PROGENITORS WITHIN CLONAL CML POPULATIONS. 189
Importin subunit beta‐1 mediates ERK5 nuclear translocation, and its inhibition synergizes with ERK5 kinase inhibitors in reducing cancer cell proliferation 188
THE ACTIN CYTOSKELETON AS A NOVEL TARGET OF AMP-ACTIVATED PROTEIN KINASE (AMPK) IN HEPATIC STELLATE CELLS 187
AML1/ETO sensitizes via TRAIL acute myeloid leukemia cells to the pro-apoptotic effects of hypoxia 184
Glucose availability in hypoxia regulates the selection of chronic myeloid leukemia progenitor subsets with different resistance to imatinib-mesylate 183
Cytoskeletal rearrangement by the mitogen-activated protein kinase ERK5 interferes with the motile and invasive phenotype of hepatocellular carcinoma cells 180
Severe hypoxia defines heterogeneity and selects highly immature progenitors within clonal erythroleukemia cells. 179
ACTIVATION OF THE MITOGEN-ACTIVATED PROTEIN KINASE ERK5 REGULATES THE DEVELOPMENT AND GROWTH OF HEPATOCELLULAR CARCINOMA 177
CXCR7 contributes to the aggressive phenotype of cholangiocarcinoma cells 177
Glutamine availability controls bcr/abl protein expression and functional phenotype of chronic myeloid leukemia cells endowed with stem/progenitor cell potential 176
Fatty acids regulate the biology of cholangiocarcinoma cells 175
Enhanced Antitumoral Activity and Photoacoustic Imaging Properties of AuNP-Enriched Endothelial Colony Forming Cells on Melanoma 172
Selective anti-leukaemic activity of low-dose histone deacetylase inhibitor ITF2357 on AML1/ETO-positive cells 171
The involvement of a Nanog, Klf4 and c-Myc transcriptional circuitry in the intertwining between neoplastic progression and reprogramming 170
Antitumor Activity of a Novel Fibroblast Growth Factor Receptor Inhibitor for Intrahepatic Cholangiocarcinoma 170
Prostaglandin E2 transactivates the colony-stimulating factor-1 receptor and synergizes with Colony-stimulating factor-1 in the induction of macrophage migration via the mitogen-activated protein kinase ERK1/2 168
HYPOXIA SELECTS DRUG-INSENSITIVE PROGENITORS WITHIN CLONAL LEUKAEMIA CELL POPULATIONS 167
Ganglioside composition correlates with the malignant phenotype of cholangiocarcinoma cells and modulates cell adhesion 165
Extracellular signal-regulated kinase 5 regulates the malignant phenotype of cholangiocarcinoma cells 164
Time- and residue-specific differences in histone acetylation induced by VPA and SAHA in AML1/ETO positive leukemia cells 162
ERK5/BMK1 is indispensable for optimal colony-stimulating factor 1 (CSF-1)-induced proliferation in macrophages in a Src-dependent fashion. 161
ACTIVATION OF ERK5, A MEMBER OF THE MAPK FAMILY, DIFFERENTIALLY REGULATES PROLIFERATION AND MIGRATION OF HEPATIC STELLATE CELLS 155
Inhibition of ERK5 elicits cellular senescence in melanoma via the cyclin-dependent kinase inhibitor p21 154
Macrophage MerTK promotes profibrogenic cross-talk with hepatic stellate cells via soluble mediators 152
Interleukin-4 rapidly down-modulates the macrophage colony-stimulating factor receptor in murine macrophages. 150
Interleukin 2 down-modulates the macrophage colony-stimulating factor receptor in murine macrophages. 150
HYPOXIA SUPPRESSES BCR/ABL AND SELECTS IMATINIB-INSENSITIVE PROGENITORS WITHIN CLONAL CML POPULATIONS. 149
ERK5 IS OVEREXPRESSED IN EXPERIMENTAL LIVER INJURY AND IN HEPATOCELLULAR CARCINOMA (HCC) AND IS MODULATED BY SOLUBLE FACTORS IN HCC CELLS 149
Mitogen-activated protein kinases and Hedgehog-GLI signaling in cancer: A crosstalk providing therapeutic opportunities? 148
Incubation of murine bone marrow cells in hypoxia ensures the maintenance of marrow-repopulating ability together with the expansion of committed progenitors. 148
EPIGENETIC MODIFICATIONS IN A T(8;21) ACUTE MYELOID LEUKAEMIA CELL LINE CAUSED BY ADMINISTRATION OF TWO HDAC INHIBITORS, VPA AND SAHA 148
c-Rel AND p27Kip ARE DOWNSTREAM TARGETS OF ERK5 IN HEPATOCELLULAR CARCINOMA (HCC) IN VIVO AND IN VITRO 146
CYTOSKELETAL REARRANGEMENT BY THE MITOGEN-ACTIVATED PROTEIN KINASE ERK5 INTERFERES WITH THE MOTILE AND INVASIVE PHENOTYPE OF HEPATOCELLULAR CARCINOMA CELLS 145
Lactate maintainsBCR/Abl expression and signalinginChronic Myeloid Leukemia cells under nutrient restriction 145
Low Mr Phosphotyrosine Protein Phosphatase Associates and Dephosphorylates p125 Focal Adhesion Kinase, Interfering with Cell Motility and Spreading 144
The Hedgehog-GLI Pathway Regulates MEK5-ERK5 Expression and Activation in Melanoma Cells 142
Altered fatty acid metabolism rewires cholangiocarcinoma stemness features 141
ERK5 mediates pro-tumorigenic phenotype in non-small lung cancer cells induced by PGE2 141
Playing the Whack-A-Mole Game: ERK5 Activation Emerges Among the Resistance Mechanisms to RAF-MEK1/2-ERK1/2- Targeted Therapy 141
Pathophysiological Impact of the MEK5/ERK5 Pathway in Oxidative Stress 140
The c-Jun-N-terminal-Kinase inhibitor SP600125 enhances the butyrate derivative D1-induced apoptosis via caspase 8 activation in Kasumi 1 t(8;21) acute myeloid leukaemia cells. 140
Salarin C inhibits maintenance of hypoxia-selected Chronic Myeloid Leukemia progenitor cells 140
GLEEVEC-RESISTANT CML STEM AND PROGENITOR CELLS ARE SENSITIVE TO HIF-INHIBITING DRUGS 138
Constitutive activation of the MAPK pathway mediates v-fes-induced mitogenesis in murine macrophages. 137
CHARACTERIZATION OF SIGNALS LINKING THE MACROPHAGE COLONY-STIMULATING FACTOR RECEPTOR TO THE FOCAL ADHESION KINASE 134
GLUCOSE AVAILABILITY IN HYPOXIA REGULATES THE SELECTION OF CML PROGENITOR SUBSETS WITH DIFFERENT RESISTANCE TO IMATINIB-MESYLATE 134
Hepatocyte-specific deletion of HIF2α prevents NASH-related liver carcinogenesis by decreasing cancer cell proliferation 133
Low M(r) phosphotyrosine protein phosphatase activity on fibroblast growth factor receptor is not associated with enzyme translocation. 131
Sphingosine 1-phosphate elicits a ROS-mediated proinflammatory response in human endometrial stromal cells via ERK5 activation 129
Opposite effects of different doses of MCSF on ERK phosphorylation and cell proliferation in macrophages. 129
Possible mechanisms and function of nuclear trafficking of the colony-stimulating factor-1 receptor. 129
Tryptophan availability selectively limits NO-synthase induction in macrophages. 128
Targeting chronic myeloid leukemia stem cells with ERK5 pathway inhibitors 128
The profile on intracellular gangliosides correlates with the malignant phenotype of cholangiocarcinoma cells and modulates cells adhesion 128
ERK5 differentially regulates PDGF-induced proliferation and migration of hepatic stellate cells. 125
Butyrates as single drug induce histone acetylation and granulocytic maturation: possible selectivity on core binding factor-acute myeloid leukemia blasts. 124
ENERGY RESTRICTION REGULATES BCR/ABL EXPRESSION IN CHRONIC MYELOID LEUKAEMIA CELLS VIA TRANSCRIPTIONAL/POST-TRANSCRIPTIONAL CONTROL 124
CSF1 Determines CSF1R Cytoplasmic Nuclear Shuttling in Breast Cancer Cells 123
MAPKS ARE INVOLVED IN THE SURVIVAL AND DIFFERENTIATION OF A T(8;21) ACUTE MYELOID LEUKEMIA CELL LINE AND ARE MODULATED BY THE HDAC INHIBITOR D1 122
ERK5-INHIBITION AS A NOVEL APPROACH TO TARGET CML STEM CELLS 122
Lipid rafts: integrated platforms for vascular organization offering therapeutic opportunities. 122
Focal adhesion kinase is redistributed to focal complexes and mediates cell spreading in macrophages in response to M-CSF. 121
SPECIFIC DEGRADATION OF THE AML1-ETO/COREPRESSOR COMPLEX BY A NOVEL ORAL HYDROXAMIC ACID DERIVATIVE 120
The low-molecular-weight phosphotyrosine protein phosphatase, when overexpressed, reduces the mitogenic response to macrophage colony-stimulating factor and tyrosine phosphorylation of its receptor. 118
INCUBATION OF MURINE ERYTHROLEUKEMIA CELLS IN SEVERE HYPOXIA INDUCES MASSIVE APOPTOSIS PARALLELED BY AKT AND ERK5 CLEAVAGE 118
Bortezomib Specifically Targets a Subset of Hypoxia-Selected CML Stem Cells 118
ERK5 inhibition elicits cellular senescence in human melanoma cells 118
BORTEZOMIB SPECIFICALLY TARGETS A SUBSET OF HYPOXIA-SELECTED CML STEM CELLS 117
HDAC INHIBITORS IN COMBINATION WITH HYPOMETHYLATING AGENTS INDUCE SPECIFIC CHROMATIN MODIFICATION AND TRANSCRIPTIONAL ACTIVATION IN CBF/AML CELLS 116
Transmodulation of cell surface regulatory molecules via ectodomain shedding. 114
EPIGENETIC MODIFICATIONS IN A T(8;21) ACUTE MYELOID LEUKEMIA CELL LINE CAUSED BY ADMINISTRATION OF THE HDAC INHIBITOR D1 114
ACETYLATION INDUCED BY BUTYRATES IN PATIENTS WITH ACUTE MYELOID LEUKEMIA CAN LEAD TO DIFFENTIATION 108
L’oncogene fes e la fosfotirosino-fosfatasi citosolica a basso peso molecolare interferiscono con la proliferazione e l’adesione cellulare 108
Glucose Availability in Hypoxia Regulates the Selection of CML Progenitor Subsets with Different Resistance to Imatinib-Mesylate 108
Totale 17.693
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Categoria #
all - tutte 51.354
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 51.354
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2020/2021911 0 0 0 0 0 164 77 109 143 205 83 130
2021/2022940 59 66 44 56 50 54 49 54 68 59 140 241
2022/20232.234 189 366 125 159 155 382 299 142 284 16 67 50
2023/2024886 30 84 122 35 58 114 54 200 25 39 86 39
2024/20256.099 218 581 355 897 1.800 826 94 344 285 219 248 232
2025/20263.461 769 860 491 493 802 46 0 0 0 0 0 0
Totale 19.398