PIRODDI, NICOLETTA
 Distribuzione geografica
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Continente #
NA - Nord America 6.605
EU - Europa 5.966
AS - Asia 2.426
SA - Sud America 344
OC - Oceania 51
AF - Africa 43
Continente sconosciuto - Info sul continente non disponibili 2
Totale 15.437
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Nazione #
US - Stati Uniti d'America 6.546
PL - Polonia 2.977
RU - Federazione Russa 1.381
SG - Singapore 630
HK - Hong Kong 578
CN - Cina 532
IT - Italia 411
IE - Irlanda 315
BR - Brasile 289
SE - Svezia 258
KR - Corea 213
VN - Vietnam 185
DE - Germania 161
FI - Finlandia 116
UA - Ucraina 99
JO - Giordania 92
GB - Regno Unito 73
IN - India 60
CH - Svizzera 56
ID - Indonesia 53
AU - Australia 45
CA - Canada 41
BE - Belgio 30
FR - Francia 30
AR - Argentina 21
CI - Costa d'Avorio 18
NL - Olanda 17
JP - Giappone 15
TR - Turchia 14
BD - Bangladesh 13
MX - Messico 11
ES - Italia 10
ZA - Sudafrica 10
EC - Ecuador 9
IQ - Iraq 9
CO - Colombia 8
RO - Romania 7
LT - Lituania 6
NZ - Nuova Zelanda 6
PE - Perù 6
SC - Seychelles 5
UZ - Uzbekistan 5
AT - Austria 4
PK - Pakistan 4
PY - Paraguay 4
TW - Taiwan 4
CZ - Repubblica Ceca 3
IR - Iran 3
MA - Marocco 3
TN - Tunisia 3
VE - Venezuela 3
AE - Emirati Arabi Uniti 2
BO - Bolivia 2
EG - Egitto 2
EU - Europa 2
GR - Grecia 2
KZ - Kazakistan 2
MY - Malesia 2
OM - Oman 2
RS - Serbia 2
SI - Slovenia 2
AM - Armenia 1
BW - Botswana 1
CL - Cile 1
DK - Danimarca 1
DO - Repubblica Dominicana 1
DZ - Algeria 1
EE - Estonia 1
GE - Georgia 1
HN - Honduras 1
HT - Haiti 1
HU - Ungheria 1
IL - Israele 1
JM - Giamaica 1
LU - Lussemburgo 1
MN - Mongolia 1
NO - Norvegia 1
NP - Nepal 1
PA - Panama 1
PH - Filippine 1
PT - Portogallo 1
QA - Qatar 1
SA - Arabia Saudita 1
SV - El Salvador 1
TT - Trinidad e Tobago 1
UY - Uruguay 1
Totale 15.437
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Città #
Warsaw 2.976
Santa Clara 1.600
Fairfield 634
Ashburn 535
Hong Kong 503
Singapore 410
Chandler 360
Woodbridge 328
Dublin 303
Seattle 276
Cambridge 246
Wilmington 212
Seoul 209
Houston 193
Beijing 191
Ann Arbor 179
Florence 136
Jacksonville 136
Altamura 95
Buffalo 95
Princeton 89
Lawrence 86
Hefei 81
Los Angeles 79
Boston 70
Munich 66
Dong Ket 64
Boardman 58
Bern 56
Jakarta 48
Ho Chi Minh City 47
San Diego 46
Dallas 45
Moscow 45
Melbourne 43
Mumbai 42
New York 41
Shanghai 39
Medford 32
Redondo Beach 31
Brussels 29
Phoenix 28
Kent 27
Turku 25
São Paulo 23
Norwalk 21
Chicago 19
Hanoi 19
Abidjan 18
Redwood City 18
Clifton 17
Rome 17
Helsinki 16
Toronto 16
Falls Church 14
Milan 13
Rio de Janeiro 13
Tokyo 13
Andover 12
Falkenstein 12
London 12
Belo Horizonte 11
Dearborn 11
Guangzhou 10
Hillsboro 10
Council Bluffs 9
Lappeenranta 9
Salt Lake City 9
Biên Hòa 8
Ottawa 8
Siena 8
Tukwila 8
Bologna 7
Frankfurt am Main 7
Paris 7
The Dalles 7
Amsterdam 6
Campinas 6
Frankfurt Am Main 6
San Jose 6
Tampa 6
Auburn Hills 5
Auckland 5
Badia Polesine 5
Bengaluru 5
Göttingen 5
Johannesburg 5
Saronno 5
Scarborough 5
Stockholm 5
Tianjin 5
West Jordan 5
Wichita 5
Yubileyny 5
Brasília 4
Brooklyn 4
Cagliari 4
Curitiba 4
Goiânia 4
Heidelberg 4
Totale 11.355
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Nome #
Atrial Remodeling in Hypertrophic Cardiomyopathy 360
Pathogenesis of hypertrophic cardiomyopathy is mutation rather than disease specific: A comparison of the cardiac troponin T E163R and R92Q mouse models 310
Deleting exon 55 from the nebulin gene induces severe muscle weakness in a mouse model for nemaline myopathy 308
Ablation of palladin in adult cardiac muscle causes cardiac dilation and systolic dysfunction 292
Myocardial Dysfunction in Hypertrophic Cardiomyopathy: Primary Effects of Sarcomeric Mutations Versus Secondary EC-Coupling Remodelling 283
Altered Cross Bridge Kinetics in Skeletal Myofibrils from NEBDex55, a Novel Mouse Model of Nebulin-Based Nemaline Myopathy 280
The familial hypertrophic cardiomyopathy-associated myosin mutation R403Q accelerates tension generation and relaxation of human cardiac myofibrils 279
Deleting exon 55 from the nebulin gene induces severe muscle weakness in a mouse model for nemaline myopathy 277
Mutations in the central part of a Tropomyosin molecule alter Ca2+ sensitivity and tension relaxation of skeletal muscle myofibrils after troponin–tropomyosin removal and reconstitution 260
Contractile effects of the exchange of cardiac troponin for fast skeletal troponin in rabbit psoas single myofibrils 254
C-Terminal Truncation of Troponin I and Substitutions of Non- Canonical Residues in the Central Part of Tropomyosin 1.1 Disrupt Thin Filament Switched Off State in Rabbit Psoas Myofibrils 253
Force regulation by Ca2+ in skinned single cardiac myocytes of frog. 251
Effect of troponin Ca2+ binding properties on the kinetics of myofibril force initiation and relaxation 250
The homozygous K280N troponin T mutation alters cross-bridge kinetics and energetics in human HCM 249
Mechanical and Energetic Consequences of HCM-Causing Mutations 248
Susceptibility of isolated myofibrils to in vitro glutathionylation: potential relevance to muscle functions 247
Calcium dependence of the apparent rate of force generation in single frog and rabbit skeletal muscle myofibrils activated by rapid solution changes 245
Active and passive forces of isolated myofibrils from cardiac and fast skeletal muscle fibres of the frog. 234
The Relaxation Properties of Myofibrils Are Compromised by Amino Acids that Stabilize α-Tropomyosin 231
A Novel Method of Isolating Myofibrils From Primary Cardiomyocyte Culture Suitable for Myofibril Mechanical Study 228
Tropomyosin flexibility modulates Ca2+ sensitivity of thin filament and affects tension relaxation in skeletal muscle myofibrils after troponin-tropomyosin removal and reconstitution 217
Design of muscle contraction assist devices by liquid crystalline elastomers 216
Increased tension cost in human familial hypertrophic cardiomyopathy caused by the MYH7 mutation R403Q 212
Relaxation kinetics following sudden Ca(2+) reduction in single myofibrils from skeletal muscle 210
Mechanical measurements from single frog skeletal muscle myofibrils activated by rapid solution changes 209
Development of Light-Responsive Liquid Crystalline Elastomers to Assist Cardiac Contraction 208
The effects of phosphate on the contraction of single skeletal myofibrils 202
Advanced Morpho-Functional Analysis on Ventricular and Atrial Tissue Reveals Cross-Bridge Kinetics Alterations and Sarcomere Energetic Impairment in Hcm Patients 195
The Missense E258K-MyBP-C Mutation Increases the Energy Cost of Tension Generation in Both Ventricular and Atrial Tissue from HCM Patients 191
The missense E258 K-MyBP-C mutation increases the energy cost of tension generation in both ventricular and atrial tissue from HCM patients 182
EFFECTS OF MAVACAMTEN, A FIRST-IN-CLASS INHIBITOR OF SARCOMERIC MYOSINS, ON THE MECHANICS OF ATRIAL AND VENTRICULAR MAMMALIAN MYOCARDIUM AND FAST SKELETAL MUSCLE 180
Impact of Mavacamten on Force Generation in Single Myofibrils from Rabbit Psoas and Human Cardiac Muscle 179
Ablation of palladin in adult heart causes dilated cardiomyopathy associated with intercalated disc abnormalities 178
ATPase and shortening rates in frog fast skeletal myofibrils by time-resolved measurements of protein-bound and free Pi. 178
The relation between sarcomere energetics and the rate of isometric tension relaxation in healthy and diseased cardiac muscle 174
New techniques in linear and non-linear laser optics in muscle research. 168
Myocardial overexpression of ANKRD1 causes sinus venosus defects and progressive diastolic dysfunction. 164
Extraction and Replacement of the Tropomyosin-Troponin Complex in Isolated Myofibrils. 163
Myosin isoform-dependent effect of omecamtiv mecarbil on the regulation of force generation in human cardiac muscle 161
Tension generation and relaxation in single myofibrils from human atrial and ventricular myocardium. 155
Impaired diastolic function after exchange of endogenous troponin I with C-terminal truncated troponin I in human cardiac muscle 155
Design of Biocompatible Liquid Cristal Elastomers Reproducing the Mechanical Properties of Human Cardiac Muscle 154
Slower Calcium Handling Balances Faster Cross-Bridge Cycling in Human MYBPC3 HCM 153
Characterization of the cross-bridge force-generating step using inorganic phosphate and BDM in myofibrils from rabbit skeletal muscles 153
Evidence that phosphate release is the rate-limiting step on the overall ATPase of psoas myofibrils prevented from shortening by chemical cross-linking 150
Mutations in MYH7 reduce the force generating capacity of sarcomeres in human familial hypertrophic cardiomyopathy. 147
The HCM-associated cardiac Troponin T mutation K280N increases the energetic cost of tension generation in human cardiac myofibrils 145
Impact of tropomyosin isoform composition on fast skeletal muscle thin filament regulation and force development 145
Time resolved depression of isometric force by Mavacamten in single myofibrils from rabbit psoas and human cardiac muscle 145
Myocardial overexpression of ANKRD1 affects developmental cardiac remodeling and leads to adult diastolic dysfunction 143
No direct effect of creatine phosphate on the cross-bridge cycle in cardiac myofibrils 142
HDAC6 contributes to pathological responses of heart and skeletal muscle to chronic angiotensin-II signaling. 142
Faster cross-bridge detachment and increased tension cost in human hypertrophic cardiomyopathy with the R403Q MYH7 mutation 142
Calcium binding kinetics of troponin C strongly modulate cooperative activation and tension kinetics in cardiac muscle 141
Myofilament calcium sensitivity does not affect cross-bridge activation-relaxation kinetics 140
Mavacamten depresses human atrial contractility in the same EC50% range as human ventricle 139
Is fast cross-bridge detachment a common feature to MYH7 and MYBP3 FHC-associated mutations in human cardiac myofibrils? 138
Functional imaging of skeletal muscle fiber in different physiological states by Second Harmonic Generation 137
Functional impact of TnT mutations responsible for Hypertrophic Cardiomyopathy on Tn-exchanged single myofibrils 135
The HCM-associated cardiac Troponin T mutation K280N accelerates tension generation and relaxation in human cardiac myofibrils 135
Contraction and electrophysiological abnormalities in myofilament mutation-positive and mutation-negative human HCM myocardium 133
Tropomyosin Ser-283 pseudo-phosphorylation slows myofibril relaxation. 132
Myopalladin is upregulated in dilated cardiomyopathies patients and myopalladin knockout mice develop cardiac dilation and dysfunction following pressure overload 132
EFFECTS OF CHRONIC ATRIAL FIBRILLATION ON ACTIVE AND PASSIVE FORCE GENERATION IN HUMAN ATRIAL MYOFIBRILS 131
Study of skeletal muscle cross-bridge population dynamics by Second Harmonic Generation 131
The effect of inorganic phosphate on force generation in single myofibrils from rabbit skeletal muscle 126
Myopalladin knockout mice develop cardiac dilation and show a maladaptive response to mechanical pressure overload 125
Genotype-Driven Pathogenesis of Atrial Fibrillation in Hypertrophic Cardiomyopathy: The Case of Different TNNT2 Mutations 125
Age-related decline in contractile function of ventricular myofibrils from HCM patients carrying MYBPC3 mutations 123
Thin filament Ca2+ binding properties and regulatory unit interactions alter kinetics of tension development and relaxation in rabbit skeletal muscle 119
The HCM-Associated Cardiac Troponin T Mutation K273N Accelerates Tension Generation and Relaxation in Human Cardiac Myofibrils 117
Effects of removal and reconstitution of troponin and tropomyosin on Ca2+-activated tension generation in cardiac myofibrils 117
Mavacamten has a differential impact on force generation in myofibrils from rabbit psoas and human cardiac muscle 117
Modulation by substrate concentration of maximal shortening velocity and isometric force in single myofibrils from frog and rabbit fast skeletal muscle 116
Functional imaging of muscle cells by Second Harmonic Generation - art. no. 60891I 112
Nebulette knockout mice have normal cardiac function, but show Z-line widening and up-regulation of cardiac stress markers 111
Endogenous troponin complex replacement in single skeletal and cardiac myofibrils 110
Calcium dependence of the apparent rate of force generation in single striated muscle myofibrils activated by rapid solution changes 109
Impact of E163R cTnT mutation on cardiac mechanics and energetics in a murine model 106
Myocardial dysfunction in hypertrophic cardiomyopathy (HCM): primary effects of sarcomeric protein mutations versus secondary E-C coupling remodeling 106
Myosin Binding Protein-C Mutations responsible for Hypertrophic Cardiomyophaty impair relaxation in human cardiac myofibrils 105
Troponin composition affects myofilament sensitivity but no cross-bridges cycle kinetics. 104
Endogenous troponin complex replacement in single skeletal and cardiac myofibrils 102
α-Tropomyosin with a D175N or E180G Mutation in Only One Chain Differs from Tropomyosin with Mutations in Both Chains. 102
MOLECULAR MECHANISMS IN CARDIAC CELL CONTRACTION AND RELAXATION 93
Novel method of obtaining myofibrils from cardiomyocyte cultures 88
Effect of TnC with altered Ca2+ binding kinetics on force generation kinetics in striated muscle 85
Tropomyosin D137L of reduced flexibility increases submaximal calcium-activated tension in skeletal muscle myofibrils after troponin-tropomyosin removal and reconstitution 83
Effect of removal and reconstitution of troponin and tropomyosin on Ca2+- activated tension generation in isolated myofibrils 83
Mechanism of cross-bridge detachment in isometric force relaxation of skeletal and cardiac muscle 81
New endogenous triggers of atrial fibrillation 81
Role of tropomyosin (Tm) isoforms in skeletal muscle thin filament regulation 77
Calcium dependence of the apparent rate of force generation in single striated muscle myofibrils activated by rapid solution changes 62
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Totale 15.616
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Categoria #
all - tutte 38.417
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 38.417
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2020/20211.090 0 0 0 0 130 158 94 133 138 256 58 123
2021/2022706 21 63 65 27 59 20 30 49 31 42 118 181
2022/20231.671 178 256 82 129 127 273 160 91 251 12 41 71
2023/2024574 29 69 99 38 34 50 38 123 10 19 36 29
2024/20254.598 169 410 238 608 1.429 612 59 401 181 105 189 197
2025/20262.148 482 590 335 357 384 0 0 0 0 0 0 0
Totale 15.616